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CCL25/CCR9 promotes the induction and function of CD103 on intestinal intraepithelial lymphocytes.

The integrin CD103 and the chemokine receptor CCR9 are co-expressed on small intestinal CD8+ intraepithelial lymphocytes (IEL), naïve murine CD8+ T cells and by a small population of effector/memory CD8+ T cells, indicating a potential role for CCR9 in regulating CD103 expression and function. Here, we demonstrate that CD103, in contrast to CCR9, is down-regulated on CD8+ T cells following their a

Onur Kilic

Forskare Kontaktinformation E-post: onur [dot] kilic [at] genus [dot] lu [dot] seOrganisation Genusvetenskap Hämtställe: 31 WebbplatsOnur Kilics profil i Lunds universitets forskningsportalBackgroundI have an interdisciplinary background in Political Science and Communication Studies and finished my master’s degree in Global Studies at Lund University in 2018. In my master’s thesis, (Re)Articulati

https://www.genus.lu.se/onur-kilic - 2026-04-30

Application admission postgraduate studies

Brevmall APPLICATION FOR ADMISSION TO POSTGRADUATE STUDIES AT THE FACULTY OF SCIENCE Subject for postgraduate studies ref no Personal details Last name, First name Personal reg. no. (yy-mm-dd-xxxx) Address Female Male Postal code, Town/City Telephone number E-mail address Citizenship Swedish Other (specify below) Former last name, if any Education Degree/courses at Lund University Higher education

https://www.science.lu.se/internal/sites/science.lu.se.internal/files/application_admission_postgraduate_studies.pdf - 2026-05-01

The 5 ' Region of the MSH2 gene involved in hereditary non-polyposis colorectal cancer contains a high density of recombinogenic sequences

MSH2 rearrangements are involved in approximately 10% of hereditary non,polyposis colorectal cancer (HNPCC) families, and in most of the rearrangements, exon I is deleted. We scanned by quantitative multiplex polymerase chain reaction (PCR) of short fluorescent fragments (QMPSF) 200 kb of genomic sequences upstream of the MSH2 transcription initiation site in 21 HNPCC families with exon I deletion