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Isolation and some properties of an IgG Fc-binding protein from group A streptococci type 15
Surgical outcomes in patients with haemophilia A or B receiving extended half-life recombinant factor VIII and IX Fc fusion proteins : Real-world experience in the Nordic countries
Interaction of the Fc part of IgG with Lancefield extracts of hemolytic streptococci. Strain specificity and activity
Ht14-kp-smmp10-2014-05-20
KP_SMMP10 _ng SMMP10 Service Management: Theory of Science and Research Fields, 15 credits Second cycle _____________________________________________________________ Confirmation The syllabus was approved by the Board of the Department of Service Management and Service Studies 20 May 2014 and valid from 1 August 2014. General information The course is part of semester 1 of the Master’s programme i
https://www.ses.lu.se/en/sites/ses.lu.se.en/files/ht14-kp-smmp10-2014-05-20.pdf - 2026-05-28
IgG Fc sialylation is regulated during the germinal center reaction upon immunization with different adjuvants
Mellanösternstudier: Samtidsfrågor i Mellanöstern
Human immunoglobulin class and subclass specificity of Fc receptors induced by herpes simplex virus type 1
Herpes simplex type 1-induced Fc receptor binds to the Cgamma2-Cgamma3 interface region of IgG in the area that binds staphylococcal protein A
Identification of the site on IgG Fc for interaction with streptococci of groups A, C and G
Interaction between herpes simplex type 1-induced Fc receptor and human and rabbit immunoglobulin G (IgG) domains
Studies of protein A and herpes simplex virus-1 induced Fc gamma-binding specificities. Different binding patterns for IgG3 from Caucasian and Oriental subjects
Biological and structural characterization of murine TRALI antibody reveals increased Fc-mediated complement activation
Transfusion-related acute lung injury (TRALI) remains a leading cause of transfusionrelated deaths. In most cases, anti-leukocyte antibodies in the transfusion product trigger TRALI, but not all anti-leukocyte antibodies cause TRALI. It has been shown that the anti-major histocompatibility complex (MHC) class I antibody 34-1-2S (anti-H-2Kd) causes TRALI in BALB/c mice (MHC class I haplotype H-2Kd)
